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Free Radical Therapy Blog » Alzheimer’s Disease

Posts Tagged ‘Alzheimer’s Disease’

Is Mercury a Cause of Alzheimer’s Disease?

Tuesday, November 30th, 2010

http://www.iaomt.org/news/archive.asp?intReleaseID=348

Many years ago (about 1984) I had the good fortune of being introduced to Dr. Tadeo Takuchi of Minamata, Japan, who was first to observe that the mysterious “Minamata Disease” affecting so many of the Minamata Bay area people was due to methyl mercury that had biotransformed from inorganic mercury being dumped into the bay from a vinyl chloride factory. What he had noticed was that the people with “brain involvement” had high levels of a particular serum marker, Beta-2-Microglobulin (B2M). Those who had the highest level in the urine, as well, were the people with the most severe brain problems. So he recommended that B2M (serum and urine) become part of my routine chemistry profiles. We’ve used it ever since with great satisfaction. Then, in 1995 OSHA adopted B2M as their multi-tier indicator for when to issue a medical advisory or remove someone from the workplace due to mercury exposure, irrespective of the chemical species of mercury.

Per Alzheimer’s Disease: It is known that B2M comprises a large portion of the shellac-like amyloid that is laid down to cover the broken microtubules that characterize the severity of dementia. Important to the Free Radical Therapy clinician: B2M is a government-approved marker for the most serious of exposures to mercury, which could be important to doctors who desire to assess mercury. At IHR, 20+ years of including B2M in our chemistry profile has confirmed that you really can’t provide a full assessment of mercury exposure and body burden without it. Too, a rise in urine B2M indicates some degree of damage to the functioning of the renal tubules where (similar to what is seen in Alzheimer’s Disease) amyloid accumulates at the site of injury so as to maintain some integrity until a degree of repair can occur.

The chemistry reveals other “footprints” of mercury exposure, which include high or low serum BUN, a reduction in fasting cortisol and G-6-PD, along with a slight rise in the MCV and the eosinophilic white blood count. A moderate rise in IgA with a slight rise in total bilirubin indicates exposure, but also indicates good genetics and better protection. A rise in Beta 2 Microglobulin in serum and/or urine indicates a highly serious circumstance.

Chemistry also provides information that demonstrates and predicts who will react to their mercury or not. Typical markers include the acid/base balance, protein status, and the level of total cholesterol. Per cholesterol, a high reading is initially protective of the brain and nerves (possibly at the expense of the heart), while a low reading leaves the brain and nerves at risk of reactivity.

Understanding how to interpret the toxic footprints of chemistry and respond to the markers provides the essence of Free Radical Therapy, whether the problem is mercury or exposure to some other toxic agent. Hopefully this information is of use to the veteran as well as anyone new to this health model-based system.

Thanks again to the IAOMT and to Consumers for Dental Choice for their dedication towards doing what they can to ban any further use of mercury in our dental fillings. Thanks also to a recent California opinion that a local (if not broader) ban on amalgam should now be imposed:

http://www.iaomt.org/news/archive.asp?intReleaseID=345

What you see at work here is the effect of “reasonable people” doing the reasonable and correct thing, allowing us all a better chance to win at life.